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1 Due to their inability to repair replication-associated errors, MSI-H/mismatch repair-deficient (MMRD) tumors harbor large numbers of single-nucleotide substitutions and frameshifts and are characterized by a high tumor mutational burden (TMB).
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Mismatch repair (MMR) deficiency is associated with tumor development and high microsatellite instability (MSI-H), and these phenotypes are observed in more than 15 different tumor types and frequently present in colon, endometrial, and gastric cancer. Correspondence to Professor Nathalie Chaput.8 Faculté de Pharmacie, Université Paris-Saclay, 91400 Orsay, France.7 Département de Biologie et pathologie médicales, Gustave Roussy, F-94805 Villejuif, France.6 Faculté de Médecine, Université Paris-Saclay, 94270 Le Kremlin-Bicêtre, France.
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5 Département de médecine oncologique, Gustave Roussy, F-94805 Villejuif, France.4 Laboratoire d'Immunomonitoring en Oncologie, Unité US-23 INSERM, UMS-3655 CNRS, Gustave Roussy, F-94805 Villejuif, France.3 INSERM-U981, Gustave Roussy, Université Paris-Saclay, F-94805 Villejuif, France.2 Département d’Innovation Thérapeutique et d’Essais Précoces, Gustave Roussy, F-94805, Villejuif, France.1 CNRS-UMR9019, Equipe labellisée Ligue Contre le Cancer, Gustave Roussy, Université Paris-Saclay, F-94805 Villejuif, France.
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